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Biomarker-Guided Antidepressants — PatSnap Eureka

Biomarker-Guided Antidepressants — PatSnap Eureka
Precision Psychiatry · Patent Intelligence

Biomarker-Guided Antidepressant Pipeline: Precision Psychiatry & Triple Reuptake Inhibitor Approaches

Major depressive disorder represents a critical unmet need, with a substantial proportion of patients failing first-line SSRI/SNRI therapies. Discover how pharmacogenomic biomarkers, triple reuptake inhibitors, and kappa-opioid antagonism are reshaping the antidepressant pipeline.

Precision Psychiatry Biomarker Modalities

Distribution of biomarker types across the antidepressant patent landscape in this dataset.

Precision Psychiatry Biomarker Modalities: Pharmacogenomic SNP panels 35%, Blood gene expression 25%, Metabolomic markers 20%, Multi-axis neuropsychiatric 12%, EEG/circadian markers 8% Distribution of biomarker modality types across the antidepressant precision psychiatry patent landscape. Pharmacogenomic SNP panels represent the largest share at 35%, followed by blood gene expression panels at 25%, derived from PatSnap Eureka patent analysis. 5 Modalities SNP panels 35% Gene expression 25% Metabolomic 20% Multi-axis 12% EEG/circadian 8%
7+
Distinct therapeutic modalities in precision psychiatry pipeline
10+
Key assignee organisations filing precision psychiatry patents
3
Neurotransmitter transporters co-inhibited by triple reuptake inhibitors
Phase 2
Clinical stage reached by liafensine (TRI) before biomarker re-stratification
Disease & Target Overview

From Trial-and-Error to Biology-Driven Prescribing

Retrieved results converge on major depressive disorder (MDD), treatment-resistant depression (TRD), and related psychiatric conditions — including bipolar disorder, suicidality, PTSD, and schizophrenia — as the primary disease contexts driving precision psychiatry innovation. The National Institute of Mental Health recognises treatment-resistant depression as a major public health challenge, underscoring the urgency of biomarker-guided approaches.

The molecular target landscape is heterogeneous, spanning multiple neurotransmitter systems. Monoamine systems remain central: retrieved patents identify serotonin (5-HT) reuptake transporters, norepinephrine transporters, and dopamine transporters as co-inhibition targets for triple reuptake inhibitors. Beyond monoamines, the kappa-opioid receptor (KOR), NMDA/glutamate pathways, vasopressin/CRH stress axis, and circadian clock gene markers all feature prominently across patent filings.

Blood gene expression biomarkers for suicidality and mood subtypes — including SAT1, FOXN3, GBP1, PIK3R5, APOL2, MARCKS, and GCOM1 — are featured prominently across Indiana University and US Department of Veterans Affairs filings. Metabolomic biomarkers (acylcarnitine C3, acylcarnitine C5, α-aminoadipate) are documented as diagnostic and treatment-monitoring tools. The PatSnap analytics platform enables deep landscape analysis across all these target classes simultaneously.

This convergence of pharmacology and molecular diagnostics is reshaping how drug developers, IP teams, and clinical researchers approach antidepressant development. Explore the full patent landscape with PatSnap Eureka to map freedom-to-operate, identify assignee strategies, and track emerging biomarker IP.

DAT/SERT/NET
Most extensively patented transporter cluster in this dataset
KOR
Validated commercial target with active Janssen patent moat
GABR-A2
AstraZeneca SNP panel predicts NMDA antagonist response
AVPR1B
SNP rs28373064 predicts V1B antagonist response (Max-Planck)
  • Pharmacogenomic SNP panels across multiple target classes
  • Blood gene expression panels for suicidality stratification
  • Metabolomic biomarkers for SSRI/SNRI efficacy monitoring
  • Circadian clock gene epigenetic markers
  • Multimodal EEG + genomic co-selection approaches
  • Gut microbiome metabolite monitoring for dynamic dosing
Therapeutic Modalities

Seven Precision Psychiatry Approaches in the Antidepressant Pipeline

From triple reuptake inhibitors with companion diagnostics to TAAR1 agonism and microbiome-guided dosing — the patent landscape reveals a field moving decisively beyond classical monoamine pharmacology.

Modality 01 · TRI + Pharmacogenomics

Triple Reuptake Inhibitors with Companion Diagnostics

DeNovo Biopharma's liafensine (DB1/DB104) simultaneously inhibits serotonin, norepinephrine, and dopamine transporters, developed to "synergize the individual, though complementary, monoaminergic treatments for depression to maximize antidepressant efficacy and tolerability." The program completed multiple Phase 2 clinical studies in TRD before discontinuation; DeNovo's IP strategy now pivots to pharmacogenomic biomarkers to identify the responsive sub-population. Euthymics' EB-1010 provides a second TRI scaffold, with Phase 2 data indicating no sexual dysfunction — a key differentiation from SSRIs.

Phase 2 completed · Biomarker re-stratification proposed
Modality 02 · KOR Antagonism

Kappa-Opioid Receptor Antagonism (Aticaprant, Janssen)

Janssen Pharmaceuticals' filings on aticaprant represent the clearest example of a fully biomarker-gated MDD therapy in this dataset. Patient eligibility requires a positive biomarker signature. The filings specifically address patients with inadequate SSRI/SNRI response, positioning aticaprant as an adjunctive or subsequent-line agent. Filings span WO, CA, and US jurisdictions (2023–2025), signaling global commercial intent. New entrants to KOR antidepressant development should conduct freedom-to-operate analysis against these biomarker-linked claim sets.

Clinical stage · Biomarker-gated enrollment
Modality 03 · NMDA/Glutamate

NMDA/Glutamate Receptor Modulators with SNP-Based Patient Selection

AstraZeneca's GABR-A2 filings focus on SNPs (rs3756007, rs11503016, rs17537359, rs1372472) in the GABA-A receptor alpha-2 subunit to predict response to NMDA antagonists including ketamine. University of Michigan filings extend pharmacogenomic decision support to NMDA antagonists, glycine receptor beta (GLRB) modulators, and AMPA receptor modulators, incorporating SNP panels and ketamine-specific topological association domain analysis. Both ketamine-derivative programs and novel NMDA-adjacent programs may benefit from licensing these SNP panels.

Preclinical/translational · Multi-SNP panels
Modality 04 · TAAR1/5-HT1A

TAAR1/5-HT1A Agonism (Ulotaront) as Augmentation

Sumitomo Pharma America's filings describe ulotaront as a trace amine-associated receptor 1 (TAAR1) and 5-HT1A agonist augmentation agent for inadequate antidepressant responders. Retrieved text describes preclinical antidepressant behavioral activity in rat models that "many conventional antidepressants are unable to treat." Its non-D2, non-classical monoamine mechanism positions it as a mechanistically novel augmentation option for treatment-resistant and refractory MDD subpopulations. A large randomized, double-blind, placebo-controlled clinical trial demonstrating efficacy in schizophrenia is referenced.

Clinical (schizophrenia) · Preclinical (MDD augmentation)
Modality 05 · Metabolomic Diagnostics

Metabolomic Biomarker Diagnostics for Depression and Treatment Monitoring

Multiple patent families (Dunlop/Jia Wei/Krishnan/Rush Medical College) cover a metabolomic biomarker suite for MDD diagnosis and treatment response monitoring, spanning acylcarnitines (C3, C5), alpha-aminoadipate, and other metabolites. These are positioned to evaluate likelihood of SSRI/SNRI response and guide treatment switching. Specific antidepressants cited include escitalopram, citalopram, fluoxetine, sertraline, vortioxetine, and duloxetine. Patents span CA, WO, IL, AU, US, and JP jurisdictions, representing a well-prosecuted academic IP campaign.

Diagnostic/translational · Multi-jurisdiction prosecution
Modality 06 · Blood Gene Expression

Blood Gene Expression Panels for Suicidality and Mood Subtypes

Indiana University Research and Technology Corporation and the US Department of Veterans Affairs have filed an extensive series of precision psychiatry patents using blood gene expression panels (SAT1, FOXN3, GBP1, PIK3R5, APOL2, MARCKS, GCOM1) to stratify suicidality risk, identify treatment candidates, and repurpose existing compounds. Algorithms combine biomarkers with clinical measures for universal and gender/diagnosis-personalized predictions. The approach is described as a "liquid biopsy" validated across multiple clinical cohorts using stepwise discovery, prioritization, and validation methodology.

Translational · Multi-cohort validated
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Identify assignee strategies, freedom-to-operate risks, and emerging biomarker IP across all seven modalities.

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Patent Landscape Data

Visualising the Biomarker-Guided Antidepressant Pipeline

Patent activity signals and therapeutic modality coverage derived from PatSnap Eureka analysis of this dataset.

Antidepressant Pipeline Modalities: Relative Patent Activity

Relative patent filing intensity across novel therapeutic approaches, from established TRI/KOR programs to emerging microbiome and circadian modalities.

Antidepressant Pipeline Modalities: TRI (High), KOR Antagonism (High), NMDA/Glutamate (Medium-High), Blood Gene Expression (Medium-High), Metabolomic Diagnostics (Medium), TAAR1 Agonism (Emerging), Microbiome Monitoring (Early) Relative patent filing activity across seven novel antidepressant therapeutic modalities in the precision psychiatry pipeline. Triple reuptake inhibitors and KOR antagonism show the highest activity levels, while microbiome and circadian approaches represent early-stage IP claims. Source: PatSnap Eureka patent analysis. High Medium Early High TRI High KOR Med-High NMDA Med-High Gene Expr. Medium Metabolom. Emerging TAAR1 Early Microbio. Source: PatSnap Eureka Patent Analysis · Precision Psychiatry Dataset

Key Assignee IP Activity: Jurisdictions & Filing Intensity

Patent filing breadth and jurisdictional coverage for the top organisations in this precision psychiatry dataset.

Key Assignee IP Activity: DeNovo Biopharma (IL/TW/MX/JP — highest intensity), Janssen Pharma (WO/CA/US — high), Indiana Univ/VA (US/WO/CA — high), Max-Planck (EP/ES — medium), Univ Michigan (JP — medium), AstraZeneca (CN/JP — medium-low), Genomind (WO/US — lower), Sumitomo (CN — emerging) Horizontal bar chart showing relative patent filing intensity and jurisdictional breadth for key organisations in the biomarker-guided antidepressant precision psychiatry patent landscape. DeNovo Biopharma leads with the most concentrated TRI/pharmacogenomics patent family. Source: PatSnap Eureka analysis. DeNovo Biopharma IL/TW/MX/JP Janssen Pharma WO/CA/US Indiana Univ / VA US/WO/CA Max-Planck EP/ES Univ. Michigan JP (2022–2025) AstraZeneca CN/JP Sumitomo CN (2025) Source: PatSnap Eureka · Precision Psychiatry Patent Dataset

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Key Molecular Targets

Molecular Target Landscape: From Transporters to Clock Genes

The precision psychiatry patent landscape spans a diverse range of molecular targets, each with distinct IP ownership and clinical development implications.

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DAT/SERT/NET Transporters

The most extensively patented pharmacological target cluster in this dataset. DeNovo Biopharma's liafensine program represents the most advanced TRI with pharmacogenomic companion diagnostics, having completed Phase 2 studies. Retrieved data confirm that genomic biomarkers are being developed post hoc to rescue clinical programs that showed insufficient efficacy on unselected populations.

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Kappa-Opioid Receptor (KOR/OPRK1)

Janssen's aticaprant filings represent a major commercial interest. The "biomarker signature positive" construct positions KOR antagonism squarely within the precision psychiatry framework for MDD with SSRI/SNRI failure. Multi-jurisdictional filings (WO/US/CA, 2023–2025) establish a strong IP position. New entrants should conduct freedom-to-operate analysis against these biomarker-linked claim sets.

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GABR-A2 & NMDAR/AMPAR/GLRB Network

AstraZeneca's GABR-A2 SNP panel (rs3756007, rs11503016, rs17537359, rs1372472) is described as predictive of response to NMDA antagonists in MDD. University of Michigan filings describe a pharmacogenomic assay incorporating SNPs, topological association domain analysis (ketamine-specific), and clinical values to stratify patients for NMDA, AMPA, or glycine receptor-based therapies in TRD.

Circadian Clock Per Gene CRE Sequence

The University of Chinese Academy of Sciences describes deletions/insertions and epigenetic modifications at the CRE sequence of the Per1/Per2 promoter as major-effect markers determining antidepressant response, linked to adenosine A1 receptor agonist CCPA's antidepressant-like action. This represents an early but active IP claim in a mechanistically novel antidepressant direction.

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Unlock 3 Additional Molecular Target Profiles
Access AVPR1B, BDNF/neuroinflammation, and TPH2/DRD2/TH polymorphism target analysis with full patent citation data.
AVPR1B SNP rs28373064 BDNF multi-analyte panel TPH2/DRD2/TH SSRI predictors
Explore Full Target Landscape in Eureka →
Combination Approaches & Emerging Directions

Seven Convergence Strategies Reshaping the Antidepressant Pipeline

Among retrieved results, multiple combination strategies and emerging directions are signalled. TRI + Pharmacogenomics: DeNovo Biopharma's filings signal a strategy of using genomic biomarker panels to identify the TRI-responsive subpopulation within the MDD continuum — a paradigm of rescuing failed broad-population trials through retrospective/prospective biomarker stratification. This model is explicitly described as replicable across other negative MDD trials in DAT/NET/SERT-targeting compounds.

KOR Antagonist + Prior SSRI/SNRI Failure as Enrichment Criterion: Janssen's aticaprant patents define the clinical combination (SSRI/SNRI inadequate response → KOR antagonist) and use biomarker signature as an enrollment gate, signaling the convergence of pharmacogenomics with adjunctive depression therapy. The PatSnap life sciences intelligence platform enables teams to track these evolving claim sets across jurisdictions in real time.

NMDA Antagonist + Multi-Receptor SNP Panel: University of Michigan filings extend beyond single-gene pharmacogenomics to multi-SNP panels combined with topological association domain analysis for ketamine-adjacent therapies, representing an emerging computational pharmacogenomics direction. The World Health Organization has identified depression as a leading cause of disability globally, reinforcing the commercial urgency of these approaches.

Gut Microbiome Metabolite Monitoring for Dynamic Antidepressant Dosing: A 2025 Chinese patent from Beijing Huilongguan Hospital describes a system integrating gut microbiota metabolite monitoring (short-chain fatty acids) with antidepressant drug metabolism kinetics for real-time dose adjustment — an emerging computational-biological convergence direction. EEG + Genomic Biomarker Co-Selection: Ortho Neuroscience's NSI-189 filings describe EEG characteristics alongside genetic biomarkers for patient selection, suggesting a multimodal precision approach combining neurophysiology with genomics. Explore the full combination strategy patent landscape via PatSnap Eureka.

Strategic Implications
  • Pharmacogenomic biomarkers are now an expected component of regulatory and commercial strategy for TRI and novel antidepressant programs, not an add-on
  • Failed broad-population Phase 2 trials may contain recoverable signals via retrospective biomarker analysis
  • KOR antagonism represents a validated commercial target with an active Janssen patent moat (WO/US/CA, 2023–2025)
  • Glutamatergic precision psychiatry is a multi-target space with differentiated IP across AstraZeneca and University of Michigan
  • TAAR1 agonism, circadian markers, and microbiome metabolites signal a shift beyond classical monoamine pharmacology
IP Team Action Items

IP teams should evaluate whether retrospective biomarker analysis of prior negative MDD trials in DAT/NET/SERT-targeting compounds could unlock value. New entrants to KOR antidepressant development should conduct freedom-to-operate analysis against Janssen's biomarker-linked claim sets.

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Clinical & Translational Signals

Pipeline Stage Summary: Key Agents in Biomarker-Guided Antidepressant Development

Clinical stage and key differentiators for the principal compounds identified in this precision psychiatry patent dataset.

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Access Full Pipeline Stage Table
View complete clinical stage data, biomarker strategies, and patent citation details for all agents in the precision psychiatry pipeline.
Liafensine Phase 2 data Aticaprant biomarker criteria NSI-189 EEG selection + more agents
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Track Every Antidepressant Patent Filing in Real Time

PatSnap Eureka monitors new filings, assignee activity, and biomarker claim sets across all jurisdictions automatically.

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Frequently asked questions

Biomarker-Guided Antidepressants — key questions answered

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References

  1. Compositions and methods for assessing the efficacy of inhibitors of neurotransmitter transporters — DeNovo Biopharma LLC, 2022 [Patent, IL]
  2. Compositions and methods for assessing the efficacy of inhibitors of neurotransmitter transporters — DeNovo Biopharma LLC, 2022 [Patent, IL]
  3. Compositions and methods for assessing the efficacy of inhibitors of neurotransmitter transporters — DeNovo Biopharma LLC, 2023 [Patent, JP]
  4. Preparation and use of (+)-1-(3,4-dichlorophenyl)-3-azabicyclo[3.1.0]hexane in the treatment of conditions affected by monoamine neurotransmitters — Euthymics Bioscience, Inc., 2012 [Patent, CA]
  5. Compositions and methods for the treatment of depression — Janssen Pharmaceuticals, Inc., 2025 [Patent, US]
  6. Compositions and methods for the treatment of depression — Janssen Pharmaceuticals, Inc., 2023 [Patent, WO]
  7. gabr-a2 diagnosis — AstraZeneca Aktiebolag, 2015 [Patent, JP]
  8. Pharmacogenomic decision support for modulators of NMDA, glycine and AMPA receptors — The Regents of the University of Michigan, 2024 [Patent, JP]
  9. Pharmacogenomic decision support for modulators of NMDA, glycine, and AMPA receptors — The Regents of the University of Michigan, 2022 [Patent, JP]
  10. Ulotaront for adjunctive treatment of major depressive disorder — Sumitomo Pharma America, Inc., 2025 [Patent, CN]
  11. Methods and compositions for diagnosing depression — Boadie W. Dunlop, 2020 [Patent, CA]
  12. Methods and compositions for diagnosing depression — Jia, Wei, 2020 [Patent, WO]
  13. Methods and compositions for diagnosing depression — Krishnan, Ranga R., 2022 [Patent, US]
  14. Blood biomarkers for suicidality — Indiana University Research & Technology Corporation, 2016 [Patent, US]
  15. Precision medicine for treating and preventing suicidality — US Department of Veterans Affairs, 2020 [Patent, US]
  16. Precision medicine for suicidality: from universality to subtypes and personalization — Niculescu et al., Marion County Coroner's Office, 2017 [Paper]
  17. Method for predicting a treatment response to a V1B antagonist — Max-Planck-Gesellschaft zur Förderung der Wissenschaften e.V., 2019 [Patent, EP]
  18. Major-effect markers influencing depressive or antidepressant behavior and their applications — University of Chinese Academy of Sciences [Patent]
  19. Biomarkers for monitoring neuropsychiatric disease treatment — Richer Diagnostics [Patent]
  20. Neuropsychiatric test reports — Genomind, LLC, 2013 [Patent, WO]
  21. Methods for treating depressed patients with poor cognition — Ortho Neuroscience, 2024 [Patent, JP]
  22. A method and device for dynamic adjustment of antidepressant dosage based on gut microbiota metabolite monitoring — Beijing Huilongguan Hospital, 2025 [Patent, CN]
  23. Concomitant medications for the treatment of depression — Nippon Medical School, 2025 [Patent, JP]
  24. Genetic polymorphism useful for predicting responsiveness to antidepressants — Shinsan Sozo Kenkyu Kiko [Patent]
  25. World Health Organization — Depression Fact Sheet
  26. National Institute of Mental Health — Treatment-Resistant Depression
  27. European Bioinformatics Institute — Pharmacogenomics Resources

All data and statistics on this page are sourced from the references above and from PatSnap's proprietary innovation intelligence platform. This report is derived from a limited set of patent and literature records retrieved across targeted searches and represents a snapshot of innovation signals within this dataset only. It should not be interpreted as a comprehensive view of the full field, clinical pipeline, or regulatory landscape.

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