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CT-388 GLP-1/GIP & Petrelintide — PatSnap Eureka

CT-388 GLP-1/GIP & Petrelintide — PatSnap Eureka
Obesity Pipeline Intelligence

CT-388 GLP-1/GIP Dual Agonist & Petrelintide: The Next Obesity Frontier

Roche's CT-388 Phase II readout and Zealand Pharma's petrelintide amylin partnership are reshaping the next generation of obesity therapeutics. Explore the patent landscape, clinical data signals, and competitive dynamics powering this inflection point.

Obesity Drug Mechanism Landscape: GLP-1 Mono, GLP-1/GIP Dual (CT-388), Amylin (Petrelintide), Triple Agonist, and Combination approaches A process diagram showing the evolution of obesity drug mechanisms from GLP-1 mono-agonism through dual GLP-1/GIP agonism (CT-388, tirzepatide), amylin receptor agonism (petrelintide), and emerging triple and combination modalities. Source: PatSnap Eureka patent and literature analysis. GLP-1 MONO Semaglutide ~15% BWL GLP-1/GIP DUAL CT-388 (Ph.II) Tirzepatide Up to 22.5% BWL AMYLIN AGONISM Petrelintide Zealand/Roche Complementary MOA TRIPLE / COMBO GLP-1/GIP/Gcg +Amylin, NPY/PYY Active investigation BRAIN PATHWAY COMPLEMENTARITY GLP-1 Pathway Area postrema Nucleus tractus solitarius Distinct neuronal populations Amylin Pathway Area postrema Lateral parabrachial nucleus Synergistic with GLP-1 agonism + Source: PatSnap Eureka · Patent & literature analysis · 2019–2024
22.5%
Max body weight loss with tirzepatide 15 mg (Phase 3)
10.8%
Weight loss with cagrilintide amylin analogue (Phase 2)
15.6%
CagriSema (amylin+GLP-1) weight loss at 20 weeks (Phase 2)
5+
Zealand Pharma amylin patent families filed since 2019
Mechanism Intelligence

Why GLP-1/GIP Dual Agonism Outperforms Mono-Agonism in Obesity

The obesity treatment landscape underwent a fundamental shift with the approval of tirzepatide, the first dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist. Phase 3 clinical trials demonstrated body weight reduction of up to 22.5% with tirzepatide 15 mg in people with obesity — a benchmark that single-mechanism GLP-1 agonists could not match. Tirzepatide represents a new class of 'twincretin' medications and has been approved for both type 2 diabetes and obesity.

CT-388, Roche's GLP-1/GIP dual agonist, is currently in Phase II clinical development targeting this same dual-receptor mechanism. Patent analysis from PatSnap analytics reveals that Chugai Pharmaceutical (a Roche subsidiary) has filed GLP-1/GIP dual agonist peptide patents covering metabolic disorders including obesity, type 2 diabetes mellitus, and non-alcoholic fatty liver disease (NAFLD). These compounds are engineered to display selectivity for both the GIP receptor and the GLP-1 receptor.

Literature analysis published in 2024 confirmed that dual GLP-1/GIP agonists reduce liver lipid droplets and alter gene expression in the liver by acting through the GLP-1 receptor, not the GIP receptor — a mechanistic insight with implications for CT-388's differentiation strategy and potential NASH label expansion. For the broader regulatory context, the FDA has been actively reviewing next-generation obesity treatments beyond GLP-1 mono-agonism.

Novo Nordisk's patent filings covering acylated GLP-1/GIP dual agonists with fatty acid linkers enabling once-weekly administration demonstrate that extended half-life formulation is a key competitive parameter in this class — a design consideration relevant to CT-388's clinical profile and commercial positioning.

22.5%
Body weight loss, tirzepatide 15 mg, Phase 3
2
Receptors targeted by CT-388: GLP-1R and GIPR
3
Indications covered: obesity, T2D, NAFLD
Ph. II
Current CT-388 clinical stage (Roche/Chugai)
  • GLP-1/GIP dual agonists show greater weight loss than GLP-1 mono-agonists
  • Tirzepatide (Eli Lilly) approved for both T2D and obesity
  • Liver effects mediated via GLP-1R, not GIPR
  • Once-weekly dosing enabled by fatty acid linker formulation
  • NAFLD/NASH potential adds label expansion opportunity
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Clinical & Patent Data Visualised

Obesity Drug Efficacy and Patent Filing Landscape

Weight loss benchmarks from published clinical data and Zealand Pharma's amylin patent filing timeline — sourced from PatSnap Eureka patent and literature analysis.

Body Weight Loss by Drug Class / Candidate

Dual GLP-1/GIP agonists achieve the highest weight loss at up to 22.5%, followed by amylin+GLP-1 combination at 15.6%, and amylin monotherapy at 10.8%.

Body Weight Loss by Drug Class: GLP-1 mono ~15%, GLP-1/GIP dual (tirzepatide) 22.5%, Amylin (cagrilintide) 10.8%, Amylin+GLP-1 (CagriSema) 15.6% Bar chart comparing percentage body weight loss across obesity drug classes derived from published Phase 2 and Phase 3 clinical trial data. Dual GLP-1/GIP agonism (tirzepatide 15 mg) leads at 22.5% BWL. Source: PatSnap Eureka literature analysis 2022–2024. 25% 19% 13% 6% 0% ~15% GLP-1 Mono 22.5% GLP-1/GIP Dual 10.8% Amylin Analogue 15.6% Amylin+ GLP-1 Combo Source: PatSnap Eureka · Published Phase 2 & 3 clinical data · 2022–2024

Zealand Pharma Amylin Patent Filing Timeline

Zealand Pharma has built a dense amylin analogue patent estate from 2019 through 2022, covering obesity treatment and synergistic combinations — underpinning petrelintide's IP position.

Zealand Pharma Amylin Patent Filing Timeline: 2019 WO2019/243491 Amylin for Obesity, 2020 WO2020 Amylin Analogues, 2021 WO2021/032792 Amylin Continuation, 2022 WO2022 Novel Peptide Analogues (x2) Timeline of Zealand Pharma's key amylin analogue patent publications from 2019 to 2022, showing progressive IP accumulation covering amylin receptor agonist analogues and their use in obesity treatment and combination therapies. Source: PatSnap Eureka patent database. 2 1.5 1 0 1 1 1 2 2019 2020 2021 2022 WO2021/032792 cited in 2022 continuations Source: PatSnap Eureka · Zealand Pharma patent database · 2019–2022

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Petrelintide & Amylin Intelligence

Zealand Pharma's Petrelintide: Amylin Agonism as Obesity Combination Backbone

Zealand Pharma's amylin analogue platform, underpinning petrelintide, is built on a deep patent estate and a scientifically validated complementary mechanism to GLP-1 agonism.

Mechanism

Distinct Brain Pathways Enable Synergistic Weight Loss

GLP-1 acts primarily in the hindbrain by activating GLP-1 receptors in the area postrema and the nucleus tractus solitarius, whereas amylin acts primarily through the area postrema and the lateral parabrachial nucleus. Both mechanisms signal via the area postrema, but activate distinct neuronal populations. The combination of amylin receptor agonism with GLP-1 receptor agonism is predicted to complement and potentially synergize to inhibit food intake, increase energy expenditure, and reduce body weight. This scientific rationale underpins Zealand's partnership strategy with major pharma partners.

Complementary neuronal activation
Patent Estate

Zealand's Amylin Patent Portfolio Spans 2019–2022

Zealand Pharma holds multiple WO patent families covering amylin analogues for obesity treatment: WO2019/243491 (amylin analogues for obesity), WO2020 (amylin analogues), WO2021/032792 (amylin analogue continuation), and two WO2022 publications titled "Novel Peptide Analogues" which explicitly reference WO2021/032792. These patents cover amylin receptor agonist analogues with improved half-life and solubility versus native human amylin, which is not therapeutically useful because of its tendency to form amyloid deposits. The patent analytics show a clear IP accumulation strategy.

5+ patent families since 2019
Combination Strategy

Synergistic Weight Reduction with GLP-1, GIP, and Other Agents

Zealand's 2019 patent (WO2019/243491) explicitly describes that a synergistic weight reducing effect may be achieved by administering an amylin analogue in combination with another agent selected from: GLP-1 agonists, GIP agonists, GLP-2 agonists, NPY receptor antagonists, PYY agonists, melanocortin receptor agonists, leptin receptor agonists, sodium glucose cotransporter (SGLT) inhibitors, or combinations thereof. This broad combination IP positions petrelintide as a versatile backbone for next-generation obesity regimens, including potential combination with CT-388. The EMA has been tracking combination obesity therapy development closely.

Broad combination IP claimed
Competitive Context

Novo Nordisk's CagriSema Sets the Combination Benchmark

Cagrilintide, Novo Nordisk's long-acting amylin analogue, exhibited dose-dependent weight loss of up to 10.8% in a Phase 2 trial. In combination with semaglutide 2.4 mg, CagriSema reduced body weight by up to 15.6% at 20 weeks in an exploratory Phase 2 trial. Amylin acts as a satiety hormone co-secreted with insulin from pancreatic beta cells, activating specific amylin receptors in the area postrema and lateral parabrachial nucleus of the brain. These results validate the amylin+GLP-1 combination rationale that Zealand's petrelintide is pursuing. New-generation amylin analogues are designed with a longer half-life and more solubility versus pramlintide (approved for diabetes). See ClinicalTrials.gov for active trial registrations.

CagriSema: 15.6% BWL at 20 weeks
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Competitive Landscape

Next-Generation Obesity Pipeline: Key Candidates and Mechanisms

A structured view of the leading obesity drug candidates, their mechanisms, assignees, and clinical stage — sourced from patent and literature analysis via PatSnap Eureka.

🔒
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See all candidates, IP assignees, clinical stages, and key differentiating data points — searchable in PatSnap Eureka.
Triple agonists NPY/PYY analogues MC4R agonists + more
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Strategic Intelligence

What the CT-388 Phase II Readout and Petrelintide Partnership Signal for the Field

Key strategic takeaways derived from patent analysis and published literature on next-generation obesity drug development.

⚗️

Dual Agonism Is the Validated Baseline — Not the Ceiling

Tirzepatide's approval established that dual GLP-1/GIP agonism outperforms GLP-1 mono-agonism. CT-388's Phase II positions Roche to compete in this validated class. But the literature is clear: many next-generation obesity treatments that engage additional metabolic hormone receptors are currently being developed, including triple agonists activating GLP-1, GIP and glucagon receptors, and amylin analogues. Dual agonism is the floor, not the ceiling, of next-generation obesity medicine.

🧬

Amylin's Complementary Neuroscience Justifies the Zealand-Roche Bet

The scientific rationale for combining amylin agonism with GLP-1 agonism is mechanistically distinct from simply adding another incretin. GLP-1 acts primarily in the hindbrain via the area postrema and nucleus tractus solitarius. Amylin acts via the area postrema and the lateral parabrachial nucleus — distinct neuronal populations. This complementarity is predicted to synergize to inhibit food intake, increase energy expenditure, and reduce body weight, justifying Zealand's petrelintide partnership strategy with Roche.

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Access the full strategic analysis including combination IP breadth and NAFLD label expansion opportunity.
Zealand IP moat analysis NAFLD/NASH opportunity + more
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Pipeline Monitoring Signals

Key Signals to Watch: CT-388, Petrelintide, and the Obesity Combination Race

Critical monitoring signals for R&D, BD, and investment teams tracking the CT-388 Phase II readout and petrelintide partnership developments.

CT-388 (Roche/Chugai) — Phase II Readout Pending
CT-388 is a GLP-1/GIP dual agonist in Phase II for obesity. Chugai Pharmaceutical's patent filings cover dual agonist peptides for metabolic disorders including obesity, T2D, and NAFLD. Phase II data will be the key catalyst for assessing differentiation from tirzepatide and CT-388's potential in the dual agonist class.
MONITORING
Petrelintide (Zealand Pharma) — Amylin Patent Estate Confirmed Active
Zealand Pharma's amylin analogue patent families (WO2019/243491, WO2021/032792, and two WO2022 "Novel Peptide Analogues" publications) confirm an active and expanding IP estate. The 2022 filings explicitly reference WO2021/032792, indicating continuation strategy. Combination claims with GLP-1, GIP, and other agents are filed.
CONFIRMED
CagriSema (Novo Nordisk) — Amylin+GLP-1 Combination Benchmark Set
Cagrilintide exhibited dose-dependent weight loss of up to 10.8% in a Phase 2 trial. In combination with semaglutide 2.4 mg, CagriSema reduced body weight by up to 15.6% at 20 weeks in an exploratory Phase 2 trial — establishing the clinical benchmark for amylin+GLP-1 combinations that petrelintide must match or exceed.
BENCHMARK SET
Triple Agonist (GLP-1/GIP/Glucagon) — Emerging Class Under Active Investigation
Multiple assignees are developing triple agonists activating GLP-1, GIP, and glucagon receptors. Additionally, neuropeptide Y receptor antagonists, peptide YY analogues, and melanocortin 4 receptor agonists are also being evaluated. This emerging multi-agonist class represents the next competitive horizon beyond CT-388 and petrelintide.
WATCH
NAFLD/NASH Label Expansion — GLP-1/GIP Dual Agonist Liver Mechanism Confirmed in Preclinical Data
2024 research confirmed dual GLP-1/GIP agonist treatment reduces liver lipid droplets and alters gene expression in the liver of diet-induced obese mice via the GLP-1 receptor, not the GIP receptor. GLP-1 and GIP receptors are also expressed in the liver. This mechanistic confirmation opens a potential NAFLD/NASH label expansion pathway for CT-388 and class peers.
OPEN OPPORTUNITY
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CT-388, Petrelintide & GLP-1/GIP Obesity Pipeline — Key Questions Answered

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References

  1. GLP-1/GIP Dual Agonist Peptides for Treatment of Metabolic Disorders — Chugai Pharmaceutical, WO patent, 2023
  2. Dual GLP-1/GIP receptor agonist treatment improves lipid droplet morphology and gene expression in the liver of obese mice via GLP-1 receptor — Published 2024
  3. Novel Peptide Analogues (Amylin Receptor Agonist Analogues) — Zealand Pharma, WO patent, 2022
  4. Novel Peptide Analogues — Zealand Pharma, WO patent, 2022
  5. GLP-1 receptor agonist and dual GLP-1/GIP receptor agonist's weight loss effects — Published 2024
  6. The next generation of obesity treatments: Beyond GLP-1 receptor agonists — Published 2024
  7. GIP Agonists and Combined GIP/GLP-1 Agonists for the Treatment of Metabolic Diseases — Novo Nordisk, WO patent, 2020
  8. Acylated GLP-1/GIP Dual Agonists — Novo Nordisk, WO patent, 2022
  9. Methods of Treating Metabolic Diseases with a GLP-1 Receptor Agonist and a GIP Receptor Agonist — Eli Lilly, WO patent, 2020
  10. Tirzepatide: A novel dual GIP and GLP-1 receptor agonist for the treatment of type 2 diabetes and obesity — Published 2022
  11. Amylin and its Analogues: A Friend or Foe for the Management of Diabetes Mellitus and Obesity? — Published 2023
  12. Amylin Analogues — Zealand Pharma, WO patent, 2021
  13. Amylin Analogues for Treatment of Obesity — Zealand Pharma, WO patent, 2019
  14. Amylin Analogues — Zealand Pharma, WO patent, 2020
  15. Combination of amylin receptor agonism and GLP-1 receptor agonism — complementary mechanisms to boost weight loss — Published 2023
  16. Cagrilintide: A Long-Acting Amylin Analogue for the Management of Obesity — Published 2022

All data and statistics on this page are sourced from the references above and from PatSnap's proprietary innovation intelligence platform.

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