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PD-1/VEGF Bispecific Antibodies in NSCLC — PatSnap Eureka

PD-1/VEGF Bispecific Antibodies in NSCLC — PatSnap Eureka
Oncology IP Intelligence

PD-1/VEGF Bispecific Antibodies in EGFR-Mutant NSCLC

Ivonescimab and a wave of competing bispecific strategies are reshaping the patent and clinical landscape for EGFR-mutant non-small cell lung cancer — a population where both checkpoint monotherapy and anti-VEGF monotherapy have historically shown limited efficacy.

Post-TKI Median PFS in EGFR-Mutant NSCLC: First-line TKIs 10–19 months, Osimertinib post-T790M 7–8 months, Platinum chemotherapy (no T790M) 4–5 months Comparison of median progression-free survival across treatment lines in EGFR-mutant NSCLC, illustrating the steep decline in efficacy after TKI failure — the clinical gap that PD-1/VEGF bispecific antibodies are designed to address. Data sourced from patent filing context (Jiang Tao, CN, 2022) via PatSnap Eureka. 20mo 15mo 10mo 5mo 0 10–19mo 1st-line TKIs 7–8mo Post-T790M 4–5mo Platinum Chemo Median PFS by Treatment Line · EGFR-Mutant NSCLC
10–19mo
Median PFS with first-line EGFR TKIs
4–5mo
Median PFS on platinum chemo post-TKI (no T790M)
87%
PFS at 6 months for amivantamab combo in treatment-naive NSCLC
≥25mo
Median DOR for amivantamab + lazertinib + chemo regimen
Disease Context

Why EGFR-Mutant NSCLC Remains a High-Unmet-Need Population

EGFR-mutant non-small cell lung cancer represents a therapeutically challenging population. EGFR TKI monotherapies — including erlotinib, gefitinib, icotinib, afatinib, osimertinib, and almonertinib — achieve median PFS of 10–19 months, but virtually all patients eventually progress. In patients without secondary T790M resistance mutations, post-TKI treatment options are largely limited to platinum-based chemotherapy with median PFS of only 4–5 months.

Anti-PD-1 monotherapy has shown poor efficacy in EGFR-mutant disease specifically — a pattern reinforced by data cited in a Regeneron Pharmaceuticals filing, which references published evidence that MET-driven NSCLC patients derive limited benefit from anti-PD-L1 monotherapy even at high tumor mutational burden. This immunosuppressive tumor microenvironment is driven by both the PD-1/PD-L1 checkpoint axis and VEGF-mediated angiogenesis — the two axes that bispecific antibodies like ivonescimab are designed to co-target in a single molecule.

The central molecular targets identified across retrieved patent results include PD-1/PD-L1, VEGF/VEGFA/VEGFR-2, EGFR (including exon 19 deletions, L858R, T790M, and exon 20 insertions), c-Met as a bypass resistance receptor, and TGF-β/TGF-βRII as an immunosuppressive cytokine. Researchers using PatSnap's IP analytics platform can map how each of these targets is claimed across competing patent families.

Primary Molecular Targets
  • PD-1 / PD-L1 — immune checkpoint axis
  • VEGF / VEGFA / VEGFR-2 — angiogenic axis
  • EGFR (exon 19 del, L858R, T790M, exon 20 ins)
  • c-Met (MET) — bypass resistance receptor
  • TGF-β / TGF-βRII — immunosuppressive cytokine
  • CD73 — emerging adenosine resistance axis
Key Assignees in Dataset
Akeso Biopharma Janssen Biotech BioNTech SE Genentech Merck Patent GmbH Jiangsu Hengrui Novartis AG Bristol Myers Squibb Pfizer Inc. Regeneron Incyte Corporation
Dataset Note

This report is derived from a limited set of patent and literature records retrieved across targeted searches. It represents a snapshot of innovation signals within this dataset only.

Therapeutic Modalities

Six Distinct Bispecific and Combination Clusters in the Pipeline

Retrieved patent results identify six converging modality clusters addressing different arms of TKI resistance and immune exclusion in EGFR-mutant NSCLC.

Modality 1 — Lead Asset

PD-1 × VEGF/VEGFA Bispecific Antibodies

Ivonescimab (Akeso Biopharma) simultaneously blocks VEGF-mediated angiogenesis and PD-1-mediated immune suppression in a single molecule. The WO filing covers advanced NSCLC, perioperative settings, HNSCC, colorectal cancer, and triple-negative breast cancer. BioNTech's 2025 WO filings enter the same space with a PD-L1-arm (not PD-1) design combined with chemotherapy and CPS ≥1 patient selection — a distinct IP claim space.

Akeso · BioNTech · WO filings
Modality 2 — Most Extensive Dataset

Bispecific EGFR/c-Met ± PD-(L)1 Inhibitors

Janssen Biotech's amivantamab (JNJ-372) patent family spans IL, EP, WO, CA, CN jurisdictions (2015–2025). Key mutations covered include L858R, T790M, exon 19 deletions, and exon 20 insertions. The bispecific format provides synergistic inhibition (14- to over 800-fold by assay) vs. monospecific mixtures. PD-(L)1 axis addition is framed as enabling ADCC and trogocytosis-based immune effector killing.

Janssen Biotech · IL/EP/WO/CA/CN
Modality 3

Anti-PD-L1 / TGF-β Bifunctional Fusion Proteins

Merck Patent GmbH's anti-PD-L1/TGFβ Trap (bintrafusp alfa) is represented across KR, IL, JP, CN filings (2021) for stage III unresectable NSCLC in treatment-naive patients, with explicit mention of EGFR-sensitizing mutation subgroups. Doses of approximately 1200–2400 mg are claimed. Jiangsu Hengrui (CN, 2022) separately claims a PD-L1 antibody fused to a TGF-βRII extracellular domain for EGFR-mutant NSCLC specifically.

Merck Patent GmbH · Jiangsu Hengrui
Modality 4

Separate PD-1 Antagonist + VEGFR Inhibitor Combinations

Genentech (CN, MX, KR, BR, 2021–2022) discloses treatment of PD-1/PD-L1 inhibitor-refractory cancers using VEGF signaling inhibitor combination, arguing that VEGF pathway co-inhibition can overcome checkpoint resistance. Pfizer's patent family (MX, ES, CN, TW, 2016–2019) covers PD-1 antagonist + VEGFR inhibitor combinations for PD-L1-expressing cancers — the pharmacological rationale that predates true bispecific formats.

Genentech · Pfizer · 2016–2022
Modality 5 — Emerging

PD-1 / LAG-3 Bispecific Antibodies in NSCLC

F. Hoffmann-La Roche's WO filing (2024) covers a PD-1-LAG-3 bispecific in NSCLC with carboplatin/paclitaxel or carboplatin/pemetrexed. Incyte's US/WO filings (2024) cover a PD-1/LAG-3 bispecific for advanced malignancies. Bristol Myers Squibb files on LAG-3 antagonist + anti-PD-1 combinations (CN, 2023) and PD-1 + CCRT + LAG-3 maintenance (CN, 2025) — indicating expansion beyond PD-1 × VEGF into dual checkpoint blockade.

Roche · Incyte · BMS · 2023–2025
Modality 6

EGFR TKI + CDK4/6 Inhibitor Combinations

Novartis AG discloses (multiple IL jurisdictions, 2020) a third-generation EGFR TKI + CDK4/6 inhibitor combination for EGFR-mutant NSCLC — a non-immunotherapy approach addressing TKI resistance via cell cycle pathway inhibition. Evidence is patent-only; development stage appears preclinical to early clinical. This modality is distinct from the PD-1/VEGF format but addresses the same post-TKI resistance problem via a different mechanism.

Novartis AG · IL · 2020
Patent Intelligence

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Data Visualisation

Clinical and Pipeline Signals from Patent Analysis

Key quantitative signals extracted from patent claim language and clinical context across the retrieved dataset.

Amivantamab Combination PFS Rates — Treatment-Naive EGFR+ NSCLC

PFS rates at multiple time points from Janssen Biotech CA filing (2025), with median DOR of at least 25 months, consistent with clinical-stage data.

Amivantamab Combination PFS Rates in Treatment-Naive EGFR-Mutant NSCLC: 87% at 6 months, 73% at 12 months, 60% at 18 months, 48% at 24 months, 41% at 30 months Progression-free survival rates decline from 87% at 6 months to 41% at 30 months for amivantamab plus lazertinib plus carboplatin plus pemetrexed in treatment-naive EGFR-positive NSCLC. Data derived from Janssen Biotech patent filing (CA, 2025) via PatSnap Eureka analysis. 100% 75% 50% 25% 0% 87% 73% 60% 48% 41% 6mo 12mo 18mo 24mo 30mo Source: Janssen Biotech CA filing (2025) via PatSnap Eureka

Therapeutic Modality Clusters by Patent Filing Activity

Relative patent filing representation across six modality clusters in the retrieved dataset, reflecting innovation intensity per approach.

Bispecific NSCLC Modality Patent Filing Activity: EGFR/c-Met Bispecific 6 families, PD-L1/TGF-β Fusion 5 families, PD-1×VEGF Bispecific 3 families, PD-1+VEGFR Combo 3 families, PD-1/LAG-3 Bispecific 3 families, EGFR TKI+CDK4/6 2 families Horizontal bar chart showing patent family representation across six therapeutic modality clusters in EGFR-mutant NSCLC, derived from PatSnap Eureka dataset analysis. EGFR/c-Met bispecifics (Janssen) lead with 6 families; PD-1×VEGF bispecifics (Akeso, BioNTech) represent 3 families. 0 2 4 6 EGFR/c-Met Bispecific 6 PD-L1/TGF-β Fusion 5 PD-1×VEGF Bispecific 3 PD-1+VEGFR Combo 3 PD-1/LAG-3 Bispecific 3 EGFR TKI+CDK4/6 2 Patent families in dataset · Source: PatSnap Eureka

Run a live patent search across all PD-1/VEGF bispecific families on PatSnap Eureka

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Assignee Landscape

Key Patent Assignees and Their Strategic Positions

Innovation activity in this dataset is predominantly patent-driven. The table below maps leading assignees to their filing geography, modality focus, and strategic signal.

🔒
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See all 11 assignees — including Bristol Myers Squibb, Pfizer, Regeneron, Novartis, Incyte, and Shanghai Junshi — with their filing jurisdictions, modality focus, and freedom-to-operate signals.
Bristol Myers Squibb Pfizer Inc. Regeneron + more
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Strategic Implications

What the Patent Landscape Signals for Drug Developers and IP Teams

Four high-priority strategic signals derived from the retrieved patent dataset for R&D strategy, competitive intelligence, and freedom-to-operate analysis.

🎯

Ivonescimab Holds First-Mover IP Position

Akeso Biopharma's WO filing covers a broad landscape of indications, combination partners, and disease stages for the anti-VEGF × anti-PD-1 bispecific format (PD-1 arm specifically). Competitors entering this space — notably BioNTech with anti-PD-L1/VEGF-A — are differentiating by targeting PD-L1 rather than PD-1, potentially circumventing Akeso's structural IP while claiming distinct combination regimen patents.

🔬

EGFR-Mutant NSCLC Is a Three-Bispecific Convergence Point

At least three distinct bispecific antibody classes converge on EGFR-mutant NSCLC in this dataset: PD-1/VEGF (ivonescimab), EGFR/c-Met (amivantamab), and PD-L1/TGF-β (bintrafusp alfa analogs). Each addresses a different arm of TKI resistance and immune exclusion. Combination strategies incorporating more than one bispecific represent a plausible but scientifically complex next frontier.

📊

Clinical PFS Data Embedded in Method-of-Treatment Claims

The Janssen Biotech EGFR/c-Met + PD-(L)1 + TKI combination data — with PFS rates embedded in claim language (87% at 6 months, declining to 41% at 30 months in treatment-naive patients) — signals that clinical-stage data are being translated into enforceable method-of-treatment claims. Drug developers and IP teams should monitor these for freedom-to-operate implications.

🇨🇳

Chinese Biopharma Filing WO Extensions Signals Global Strategy

Chinese biopharma assignees — Akeso, Jiangsu Hengrui, Shanghai Junshi, TJ Biopharma — are filing primarily in CN jurisdiction with WO extensions, indicating both domestic market protection and international patent strategy. The Jiangsu Hengrui PD-L1/TGF-βRII fusion protein for EGFR-mutant NSCLC specifically is a differentiated asset in the Chinese IP landscape that may compete with or complement ivonescimab clinically.

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Access the full analysis of biomarker-driven patient selection strategies and triple-target combination regimens shaping the next IP generation.
CD73 biomarker strategy Triple-target regimens FTO implications
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Clinical Translation

Translational Signals from Patent Claim Language

Among retrieved results, the most concrete clinical translation signals come from Janssen Biotech's amivantamab filings. A CA filing (2025) for treatment-naive patients provides specific PFS rates at multiple time points — 87% at 6 months, 73% at 12 months, 60% at 18 months, 48% at 24 months, and 41% at 30 months — with median DOR of at least 25 months. This language is consistent with data derived from completed or ongoing clinical studies embedded directly into enforceable method-of-treatment claims.

Merck Patent GmbH's TGF-β filings reference specific dose levels — 1200 mg Q2W, 1800 mg Q3W, 2400 mg Q3W — for the anti-PD-L1/TGFβ Trap in stage III NSCLC patients including those with EGFR-sensitizing mutations, signaling IND-enabling or clinical-stage dosing decisions. The ClinicalTrials.gov registry and EMA databases provide complementary clinical context for these patent signals.

For ivonescimab specifically, the Akeso WO filing's claim breadth across perioperative, advanced, and combination settings is consistent with a molecule that has progressed beyond pure preclinical development. However, no trial-level efficacy data — such as PFS or ORR — are explicitly stated within the retrieved patent text. PatSnap's life sciences intelligence platform links patent claims to clinical trial registrations for a complete development picture.

No retrieved results contain explicit Phase 3 trial outcome data, regulatory submission language, or approved product labeling for PD-1/VEGF bispecifics in EGFR-mutant NSCLC specifically. Researchers should use PatSnap Eureka alongside WHO clinical trial databases for a comprehensive regulatory picture.

Top Clinical Translation Signals
Amivantamab + Lazertinib + Chemo
PFS 87%→41% over 30 months; median DOR ≥25 months (Janssen CA, 2025)
Bintrafusp Alfa (Merck Patent GmbH)
Doses 1200mg Q2W, 1800mg Q3W, 2400mg Q3W — IND-enabling signals in EGFR-mut NSCLC subgroups
Ivonescimab (Akeso WO)
Claim breadth across perioperative + advanced NSCLC consistent with clinical-stage molecule; no PFS/ORR data in retrieved text
Jiang Tao CN Filing (2022)
Clinical data-grounded IP: cites 10–19mo TKI PFS, 7–8mo osimertinib post-T790M, 4–5mo platinum chemo without T790M
Emerging Resistance Axis

Shanghai Junshi Biosciences and TJ Biopharma (both 2025) independently filed on CD73 expression as a predictive biomarker for response to CD73 antagonist + PD-1/PD-L1 combination therapy in NSCLC — signaling CD73-mediated purinergic immune suppression as an emerging resistance axis in checkpoint-treated NSCLC populations.

Frequently asked questions

PD-1/VEGF Bispecific Antibodies in NSCLC — key questions answered

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References

  1. Antibody that binds VEGFA and PD-1 and uses thereof to treat cancers — Akeso Biopharma Co., Ltd., 2026, WO [Patent]
  2. Combination therapy comprising a bispecific anti-VEGF-A and anti-PD-L1 antibody and a chemotherapy for cancer treatment — BioNTech SE, 2025, WO [Patent]
  3. Combination therapy comprising a PD-1/PD-L1 and VEGF/VEGFR binding agent and a chemotherapy for cancer treatment — BioNTech SE, 2025, WO [Patent]
  4. Method and medicament for treating cancer unresponsive to PD-1/PD-L1 signaling inhibitor — Genentech, Inc., 2021, MX [Patent]
  5. Methods and agents for treating cancers that do not respond to PD-1/PD-L1 signaling inhibitors — Genentech, Inc., 2021, KR [Patent]
  6. Method for treating cancer unresponsive to PD-1/PD-L1 signaling inhibitor — Chinese filing — Genentech, Inc., 2022, CN [Patent]
  7. Combination of a PD-1 antagonist and a VEGFR inhibitor for treating cancer — Pfizer Inc., 2016, MX [Patent]
  8. Combined therapy of PD-1 antagonist and VEGFR-2 antagonist for treating cancer patients — Pharmaceutical Antibody Company (药物抗体公司), 2023, CN [Patent]
  9. Combination therapies with bispecific anti-EGFR/c-Met antibodies and anti-PD-1 antibodies — Janssen Biotech, Inc., 2024, WO [Patent]
  10. Use of TGF-β receptor-containing fusion protein in the preparation of drugs for treating non-small cell lung cancer — Jiangsu Hengrui Pharmaceutical Co., Ltd., 2022, CN [Patent]
  11. Use of antibody/TGF-β protein for treatment of NSCLC — Merck Patent GmbH, 2021, IL [Patent]
  12. Application of a pharmaceutical composition containing an anti-PD-1 antibody in preparing drugs for treating advanced non-small cell lung cancer — Jiang Tao, 2022, CN [Patent]
  13. Therapeutic combination of a third generation EGFR tyrosine kinase inhibitor and a cyclin D kinase inhibitor — Novartis AG, 2020, IL [Patent]
  14. National Cancer Institute — Non-Small Cell Lung Cancer Treatment (PDQ) — cancer.gov
  15. ClinicalTrials.gov — NSCLC Bispecific Antibody Trials Registry — clinicaltrials.gov
  16. World Health Organization — International Clinical Trials Registry Platform — who.int

All data and statistics on this page are sourced from the references above and from PatSnap's proprietary innovation intelligence platform. This report represents a snapshot of innovation signals within the retrieved dataset only and should not be interpreted as a comprehensive view of the full field, clinical pipeline, or regulatory landscape.

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