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PD-L1/VEGF Bispecific Antibody Pipeline — PatSnap Eureka

PD-L1/VEGF Bispecific Antibody Pipeline — PatSnap Eureka
Oncology Patent Intelligence

PD-L1/VEGF Bispecific Antibody Pipeline in Solid Tumors

Pumitamig and the broader class of PD-L1/VEGF bispecific antibodies simultaneously disable tumor immune evasion and block tumor vasculogenesis—addressing both immunological and structural barriers to durable anti-tumor responses in solid tumors. Explore the patent and literature landscape with PatSnap Eureka.

PD-L1 Inhibitor Response Rates: Unselected Patients 14–23%, PD-L1-Expressing Tumors 16–48% Comparison of response rates for approved PD-1/PD-L1 inhibitors (nivolumab, pembrolizumab, atezolizumab) in unselected vs. PD-L1-expressing patient populations, establishing the therapeutic rationale for PD-L1/VEGF bispecific antibody development. Source: WuXi Biologics patent filings via PatSnap Eureka. 50% 40% 30% 20% 10% 14% Unselected (low) 23% Unselected (high) PD-L1 selected → 16% PD-L1+ (low) 48% PD-L1+ (high) Unselected patients PD-L1-expressing tumors
14–23%
Response rate, unselected patients (approved PD-1/PD-L1 inhibitors)
16–48%
Response rate in PD-L1-expressing tumors
6+
Independent patent families articulating PD-L1/VEGF dual-targeting rationale
10+
Solid tumor indications claimed across PD-L1/VEGF patent families
Disease & Target Background

Why PD-L1 and VEGF Are Targeted Together in Solid Tumors

PD-L1 (CD274) is a type-I transmembrane protein expressed on tumor cells, T cells, B cells, dendritic cells, and macrophages. Its interaction with PD-1 (CD279) on activated T cells delivers inhibitory signals that enable tumor immune evasion. Approved PD-1/PD-L1 inhibitors—nivolumab, pembrolizumab, and atezolizumab—achieve response rates of only 14–23% in unselected patient populations, underscoring the need for augmented strategies.

VEGF (vascular endothelial growth factor) is identified across multiple retrieved patents as a key driver of tumor angiogenesis and, critically, as a mediator of immune suppression within the tumor microenvironment. Anti-VEGF therapy can restore anti-PD-1/PD-L1 activity by inhibiting VEGF-associated immunosuppression, promoting T cell infiltration into tumors, and enabling T cell priming against tumor antigens.

The scientific rationale for simultaneous PD-L1 and VEGF blockade is articulated across at least six retrieved patent families as producing superior anti-tumor efficacy compared to monotherapy. This mechanistic convergence is the foundation of the bispecific antibody programs now entering clinical development across multiple solid tumor types.

14–23%
Response rate, unselected patients
16–48%
Response rate, PD-L1-expressing tumors
6+
Patent families supporting dual PD-L1/VEGF rationale
10+
Solid tumor indications claimed
Key Indications
NSCLC HCC Gastric/GEJ RCC TNBC Bladder Ovarian Colorectal Head & Neck Melanoma
Therapeutic Modalities

Six Molecular Architectures Targeting PD-L1 and VEGF

Retrieved patent records reveal a diverse engineering landscape—from canonical bispecific antibodies to recombinant fusion proteins and VEGFR-trap domain designs—all seeking to co-block PD-L1 and VEGF in a single molecule or combination regimen.

Core Modality

PD-L1 × VEGF Bispecific Antibodies

WuXi Biologics describes a bispecific antibody that simultaneously binds human PD-L1 and VEGF with high affinity, blocking both the PD-1/PD-L1 signaling axis and VEGF/VEGFR signaling in a single molecule. This is the architecture associated with pumitamig's competitive class. First PCT application filed January 2020 (PCT/CN2020/073497), with publications through 2023–2025 across WO and JP jurisdictions.

WuXi Biologics · 4+ patent families
Fusion Protein

PD-L1/VEGF Recombinant Fusion Proteins

ImmunOnco Biopharmaceuticals (Shanghai) describes fusion proteins linking an anti-PD-L1 antibody or fragment to a VEGF-binding peptide via a linker at the N-terminus of the heavy or light chain. Claimed to achieve comparable ADCC, ADCP, and PD-1/PD-L1 and VEGF/VEGFR blockade as combination use of separate agents, with superior in vivo performance versus combination regimens.

ImmunOnco · CN & JP jurisdictions
Bifunctional Antibody

Anti-PD-L1 × VEGF Bifunctional Antibody

Huabo Biopharmaceutical Technology (Shanghai) describes bifunctional antibodies combining anti-PD-L1 with anti-VEGF elements via a linker peptide containing antibody constant region sequences, linking the anti-VEGF element to the N-terminus of the anti-PD-L1 heavy chain variable region. AP Biosciences describes a VEGF inhibitory domain (VID) fused to the Fc domain of an anti-PD-L1 antibody, with demonstrated retention of antigen-binding specificity in IFN-γ-stimulated A549 (NSCLC) and NCI-H292 cell lines.

Huabo · AP Biosciences · preclinical
VEGFR-Trap Design

Anti-VEGF/PD-L1 Bispecific with VEGFR-Trap Domain

Unimonoantibody (Beijing) Biotechnology describes a bispecific fusion protein where an anti-PD-L1 full antibody is combined with one Ig-like domain from VEGFR1 and one Ig-like domain from VEGFR2 fused to the C-terminus of the antibody heavy chains. This VEGFR trap-based design is structurally distinct from anti-VEGF antibody fragments and claimed to enable effective tumor cell killing.

Unimonoantibody · JP jurisdiction · 2024
Triple-Target

Multi-Domain Fusion Proteins (PD-L1 + VEGF + TGF-β)

Zhejiang Dao'er Biotechnology describes a triplicate fusion protein combining an anti-PD-L1 single-domain antibody fragment, a VEGF antagonist fragment, and a TGF-β binding fragment in a single molecule. The rationale is to integrate three independent immunosuppressive barriers—immune checkpoint, angiogenesis, and TGF-β-mediated immune exclusion—in a unified construct.

Zhejiang Dao'er · 2024 · next-gen
Combination Regimen

Anti-VEGF + Anti-PD-L1 Combination (Separate Agents)

Genentech describes combination regimens using a VEGF antagonist and a PD-L1 axis binding antagonist as separate agents, with preclinical evidence in mouse ovarian cancer (OV2944-HM-1) and colorectal cancer (Colon 38) models. MedImmune describes clinical dosing regimens for ramucirumab (anti-VEGFR-2) combined with durvalumab (anti-PD-L1) for gastric/GEJ adenocarcinoma, NSCLC, and HCC—with specified dosing regimens indicating active clinical trial context.

Genentech · MedImmune · clinical signals
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Patent Landscape Data

Key Data Points from the PD-L1/VEGF Patent Record

Quantitative signals extracted from retrieved patent families—assignee activity, indication breadth, and response rate context—visualised from the PatSnap Eureka dataset.

PD-L1/VEGF Patent Families by Assignee

WuXi Biologics leads with at least 4 retrieved patent families; ImmunOnco and Sanyou Biopharmaceuticals each hold 2 families in this space.

PD-L1/VEGF Patent Families by Assignee: WuXi Biologics 4, ImmunOnco 2, Sanyou 2, Biotheus 1, Huabo 1, Unimonoantibody 1 Distribution of retrieved PD-L1/VEGF bispecific antibody patent families by assignee, showing WuXi Biologics as the most active filer with 4 families across WO and JP jurisdictions (2021–2025). Source: PatSnap Eureka patent dataset. 4 3 2 1 0 4 WuXi Biologics 2 ImmunOnco 2 Sanyou 1 Biotheus 1 Huabo 1 Unimono

Indication Coverage Across PD-L1/VEGF Patent Families

NSCLC and HCC are the most frequently cited indications; broad solid tumor labeling strategies cover 10+ tumor types within the same patent families.

Indication Coverage in PD-L1/VEGF Bispecific Patents: NSCLC, HCC, Gastric/GEJ, RCC, TNBC, Bladder, Ovarian, Colorectal, Head & Neck, Melanoma — all cited within same patent families Breadth of solid tumor indications claimed across retrieved PD-L1/VEGF bispecific antibody patent families, reflecting developers' broad labeling strategies consistent with approved checkpoint inhibitors. Source: PatSnap Eureka patent dataset. NSCLC Primary HCC Primary Gastric/GEJ RCC TNBC Bladder Ovarian Colorectal +2 more 10+ solid tumor indications Broad labeling strategy across all programs Consistent with approved CPI labeling approach

PD-L1/VEGF Bispecific Molecular Architecture Landscape

Five distinct engineering formats have been patented for simultaneous PD-L1 and VEGF blockade, from canonical bispecific antibodies to VEGFR-trap fusions and triple-target constructs.

PD-L1/VEGF Molecular Architecture Landscape: Bispecific Antibody (WuXi), Fusion Protein (ImmunOnco), Bifunctional Antibody (Huabo/AP Bio), VEGFR-Trap Fusion (Unimonoantibody), Triple-Target Fusion (Zhejiang Dao'er) Five distinct molecular engineering formats patented for simultaneous PD-L1 and VEGF blockade, showing the progression from canonical bispecific antibodies to next-generation triple-target fusion proteins. Source: PatSnap Eureka patent dataset. Bispecific Antibody WuXi Biologics Fusion Protein ImmunOnco Bifunctional Ab + VID Huabo / AP Bio VEGFR Trap Fusion Unimonoantibody Triple-Target PD-L1+VEGF +TGF-β Zhejiang Dao'er Separate Combination Genentech / MedImmune NEXT-GEN

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Assignee & Competitive Landscape

Who Is Filing in the PD-L1/VEGF Bispecific Space?

Innovation is concentrated among Chinese biotech companies and large multinational pharmaceutical corporations, with activity predominantly patent-driven. Parties entering this space must conduct freedom-to-operate analysis across multiple architectural claims.

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WuXi Biologics — Lead Filer

The most prominently represented assignee for PD-L1 × VEGF bispecific antibodies specifically, with at least four patent families retrieved across WO and JP jurisdictions (2021–2025), covering the bispecific antibody structure, antigen-binding portions, manufacturing methods, and therapeutic applications. This is the assignee associated with pumitamig's competitive class.

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ImmunOnco & Biotheus — Chinese Biotech Challengers

ImmunOnco Biopharmaceuticals (Shanghai) holds retrieved patent families in both CN and JP jurisdictions (2023–2025) covering recombinant anti-PD-L1/VEGF fusion proteins. Biotheus Inc. holds a retrieved patent (CA jurisdiction, 2022) for anti-VEGF-anti-PD-L1 bispecific antibody, claiming dual immunoactivation and angiogenesis blockade.

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Roche / Genentech strategy MedImmune clinical signals University of Texas IP + more
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Clinical & Translational Signals

From Preclinical to Clinical: What the Patent Record Reveals

Retrieved results yield limited direct clinical evidence for PD-L1/VEGF bispecific antibodies as a unified molecule. The most proximate clinical signal comes from the combination-agent literature. MedImmune's retrieved patent explicitly specifies clinical dosing regimens—anti-VEGFR-2 (ramucirumab) at 8 mg/kg every two weeks combined with anti-PD-L1 (durvalumab) at 750 mg every two weeks for gastric/GEJ or HCC; 10 mg/kg every three weeks with 1125 mg anti-PD-L1 for NSCLC—indicating IND-enabling or active clinical trial context.

Genentech's retrieved patent explicitly addresses patients with "locally advanced or metastatic sarcomatoid RCC" not previously treated, suggesting clinical stage biomarker-selection methodology is in active development. A retrieved patent from Pfizer (TW jurisdiction) describes a PD-1 antagonist + VEGFR inhibitor combination specifically for PD-L1-expressing cancers, indicating clinical-stage translation of the mechanistic rationale underlying PD-L1/VEGF bispecifics.

Retrieved results do not contain pivotal trial outcome data or regulatory submission records for any single PD-L1/VEGF bispecific molecule. For comprehensive clinical pipeline analytics, PatSnap Eureka integrates patent, literature, and trial data in a unified view.

  • Anti-VEGFR-2 + anti-PD-L1: clinical dosing regimens specified for gastric/GEJ, HCC, and NSCLC
  • Sarcomatoid RCC: biomarker-selection methodology in active clinical development (Genentech)
  • PD-L1 monotherapy biomarkers: IFNG, CXCL9, CD274, and LAG3 companion signatures described (MedImmune)
  • PD-1 + VEGFR inhibitor combination: clinical-stage translation described by Pfizer for PD-L1-expressing cancers
  • No pivotal trial outcome data or regulatory submissions identified for a unified PD-L1/VEGF bispecific molecule
Clinical Dosing Signals (MedImmune)
Gastric/GEJ & HCC
Ramucirumab 8 mg/kg Q2W + Durvalumab 750 mg Q2W
NSCLC
Ramucirumab 10 mg/kg Q3W + Durvalumab 1125 mg Q3W
Biomarker Companion Strategies
PD-L1 expression (≥1% in TIICs, bladder cancer)
IFNG / CXCL9 / LAG3 gene signatures
ImmunoPET imaging (radiolabeled anti-PD-L1, Regeneron)
Sarcomatoid histology (Genentech, RCC)
PD-L2 co-expression (double ORR vs. PD-L1 alone)
Emerging Directions

Beyond Dual Targeting: Next-Generation Combination Strategies

Retrieved patent signals reveal the competitive frontier is advancing beyond PD-L1/VEGF dual-targeting toward triple-target constructs, innate immune activation, and conditionally active architectures.

Triple-Target · 2024

PD-L1 + VEGF + TGF-β Fusion Proteins

Zhejiang Dao'er Biotechnology (2024) describes multi-domain fusion proteins combining anti-PD-L1 sdAb, VEGF antagonist fragment, and TGF-β binding fragment. TGF-β immunosuppression—a documented resistance mechanism to PD-1/PD-L1 therapy—is being incorporated into next-generation multi-functional fusion designs. GlaxoSmithKline describes a related ICOS antibody + anti-PD-L1/TGF-βR fusion protein combination (JP, 2023).

Zhejiang Dao'er · GSK · resistance mechanism
Innate Immune Activation · 2024

PD-L1/CD47 Bispecific Antibodies

WuXi Biologics Ireland (WO, 2024) describes bispecific antibodies targeting PD-L1 and CD47 (the "don't eat me" signal), combining immune checkpoint blockade with innate immune activation via phagocytosis promotion. This represents a mechanistic extension of the WuXi bispecific platform beyond the VEGF axis.

WuXi Biologics · CD47 · innate immunity
Dual Checkpoint · 2023–2024

PD-L1/PD-L2 Dual Blockade

The University of Texas System (JP/CN, 2023–2024) provides preclinical xenograft data demonstrating superiority of dual PD-L1/PD-L2 bispecific antibodies over single-target anti-PD-L1 in MDA-MB-231 (TNBC) and MC38-PDL2 (colorectal) models. PD-L2-positive/PD-L1-positive patients achieve double the objective response rate compared to PD-L1-positive/PD-L2-negative patients (ChiaTai Tianqing clinical data).

University of Texas · 2× ORR in PD-L2+ patients
Conditionally Active · 2018–2023

Activatable Anti-PD-L1 Antibodies

CytomX Therapeutics (JP, 2018, 2023) describes protease-cleavable "masked" anti-PD-L1 antibodies that are activated preferentially in the tumor microenvironment, reducing systemic toxicity. This represents a precision engineering direction complementary to bispecific approaches, addressing the safety profile challenge of systemic checkpoint blockade.

CytomX · tumor microenvironment activation
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ABL Bio / MSD 4-1BB data Roche ANG-2 strategy + more signals
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Strategic Implications

What the Patent Record Signals for Developers and Investors

Five strategic implications derived from the retrieved patent dataset, relevant to parties evaluating freedom-to-operate, clinical differentiation, and pipeline durability in the PD-L1/VEGF bispecific space.

Strategic Signal Evidence from Retrieved Patent Record Implication
Highly competitive IP landscape At least five independent assignees (WuXi Biologics, ImmunOnco, Biotheus, Sanyou, Huabo) pursuing distinct molecular architectures covering the PD-L1/VEGF bispecific space, with Chinese biotech firms representing the majority of new filings. Freedom-to-operate analysis required across full bispecific antibodies, fusion proteins with VEGF-binding peptides, and VEGFR-trap domain fusions.
Mechanistic rationale is prior art Multiple independent assignees converge on the same mechanistic argument—VEGF suppresses T cell infiltration and priming, which directly undermines PD-L1 blockade—across at least six retrieved patent families. Differentiation must come from molecular format, PK/PD properties, or indication-specific biomarker strategies rather than mechanism claims.
Broad indication strategies dominate Retrieved results do not restrict claimed indications to single tumor types; NSCLC, HCC, gastric/GEJ, RCC, TNBC, colorectal, and ovarian cancers are all cited within the same patent families. Developers are pursuing broad solid tumor labeling strategies, consistent with the observed approach for approved checkpoint inhibitors.
Biomarker selection is a differentiator Genentech, MedImmune, and Regeneron show active IP development around PD-L1 expression-based patient stratification—immunoPET imaging, IFNG/CXCL9/LAG3 signatures, sarcomatoid histology—as companion strategies. Bispecific programs that co-develop predictive biomarkers will likely have stronger clinical differentiation and regulatory positioning.
Triple-target constructs are the next frontier Patents covering PD-L1 + VEGF + TGF-β and PD-L1 + VEGF + PD-L2 formats signal that the competitive frontier is advancing beyond dual-targeting. Investors and developers should evaluate pipeline durability against the multi-functional protein engineering trend now visible in the 2023–2024 filing cohort.

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References

  1. A bispecific Anti-PD-L1/VEGF antibody and uses thereof — WuXi Biologics (Shanghai) Co. Ltd., 2021, WO [Patent]
  2. A bispecific Anti-PD-L1/VEGF antibody and uses thereof — WuXi Biologics Ireland Limited, 2023, WO [Patent]
  3. Bispecific anti-PD-L1/VEGF antibodies and uses thereof — WuXi Biologics Ireland Limited, 2024, JP [Patent]
  4. Recombinant fusion protein targeting PD-L1 and VEGF, its preparation and use — ImmunOnco Biopharmaceuticals (Shanghai) Inc., 2025, JP [Patent]
  5. Recombinant fusion protein targeting PD-L1 and VEGF, its preparation and use — ImmunOnco Biopharmaceuticals (Shanghai) Inc., 2023, CN [Patent]
  6. Anti-VEGF-Anti-PD-L1 bispecific antibody, pharmaceutical composition of same, and uses thereof — Biotheus Inc., 2022, CA [Patent]
  7. Anti-PD-L1/VEGF bifunctional antibodies and their uses — Huabo Biopharmaceutical Technology (Shanghai) Co., Ltd., 2023, JP [Patent]
  8. Bifunctional proteins combining checkpoint blockade for targeted therapy — AP Biosciences Inc., 2021, JP [Patent]
  9. Bispecific fusion proteins — Unimonoantibody (Beijing) Biotechnology Co., Ltd., 2024, JP [Patent]
  10. Multidomain fusion proteins and their applications — Zhejiang Dao'er Biotechnology Co., Ltd., 2024, JP [Patent]
  11. Methods and drugs for treating cancer non-responsive to PD-1/PD-L1 signaling inhibitors — Genentech, Inc., 2022, CN [Patent]
  12. Combination of anti-VEGFR-2 and anti-PD-L1 antibodies for the treatment of cancer — MedImmune Limited, 2022, JP [Patent]
  13. Diagnostic and therapeutic methods for sarcomatoid kidney cancer — Genentech, Inc., 2025, JP [Patent]
  14. Combination therapy of an antibody that binds to angiopoietin 2 with an antibody that binds to programmed death ligand 1 — F. Hoffmann-La Roche AG, 2018, JP [Patent]
  15. Bispecific antibodies against human PD-L1 and PD-L2 and methods of use thereof — Board of Regents, The University of Texas System, 2024, JP [Patent]
  16. Bispecific antibodies against PD-L1 and CD47, method for preparing the same, and use thereof — WuXi Biologics Ireland Limited, 2024, WO [Patent]
  17. Anti-PD-L1/Anti-4-1BB bispecific antibodies and uses thereof — ABL Bio Inc., 2021, SG [Patent]
  18. Radiolabeled anti-PD-L1 antibodies for immunoPET imaging — Regeneron Pharmaceuticals, Inc., 2022, JP [Patent]
  19. Compositions and methods for characterizing the response of solid tumors to anti-PD-L1 antibody monotherapy — MedImmune, LLC, 2019, JP [Patent]
  20. Liver Cancer (HCC) — National Cancer Institute
  21. CD274 (PD-L1) Gene — NCBI Gene Database
  22. Oncology Guidelines — European Medicines Agency (EMA)

All data and statistics on this page are sourced from the references above and from PatSnap's proprietary innovation intelligence platform. This report is derived from a limited set of patent and literature records retrieved across targeted searches and represents a snapshot of innovation signals within this dataset only.

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